{"id":36406,"date":"2026-03-30T19:42:55","date_gmt":"2026-03-30T14:12:55","guid":{"rendered":"https:\/\/atsixty.com\/?p=36406"},"modified":"2026-04-07T00:32:32","modified_gmt":"2026-04-06T19:02:32","slug":"cvs-pathology","status":"publish","type":"post","link":"https:\/\/atsixty.com\/index.php\/2026\/03\/30\/cvs-pathology\/","title":{"rendered":"CVS Pathology"},"content":{"rendered":"\n<div id=\"atsixty-cvs-quiz\">\n<style>\n#atsixty-cvs-quiz {\n  --terra: #C0603A;\n  --terra-light: #d4795a;\n  --terra-pale: #fdf3ef;\n  --teal: #2A7A6F;\n  --teal-light: #3a9688;\n  --teal-pale: #eef6f5;\n  --ink: #1e1e1e;\n  --muted: #6b6b6b;\n  --rule: #e2d8d4;\n  --correct-bg: #e8f5e9;\n  --correct-border: #43a047;\n  --wrong-bg: #fdecea;\n  --wrong-border: #e53935;\n  --white: #fff;\n  --shadow: 0 2px 16px rgba(192,96,58,0.08);\n  font-family: Georgia, 'Times New Roman', serif;\n  color: var(--ink);\n  margin: 1rem 0 2rem;\n}\n#atsixty-cvs-quiz * { box-sizing: border-box; margin: 0; padding: 0; }\n\n#atsixty-cvs-quiz .quiz-shell {\n  background: #f7f2ee;\n  border: 1px solid #eee3dd;\n  border-radius: 12px;\n}\n#atsixty-cvs-quiz .quiz-sentinel { height: 1px; 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}\n#atsixty-cvs-quiz .submit-btn.visible { display: block; animation: cvsSlideUp 0.3s ease; }\n\n@keyframes cvsFadeIn  { from { opacity:0; transform:translateY(-4px); } to { opacity:1; transform:translateY(0); } }\n@keyframes cvsSlideUp { from { opacity:0; transform:translateX(-50%) translateY(12px); } to { opacity:1; transform:translateX(-50%) translateY(0); } }\n<\/style>\n\n<div class=\"quiz-shell\">\n  <div class=\"quiz-sentinel\" id=\"cvs-sentinel\"><\/div>\n\n  <div class=\"status-bar\" id=\"cvs-status-bar\">\n    <div class=\"progress-wrap\"><div class=\"progress-bar\" id=\"cvs-progress\"><\/div><\/div>\n    <div class=\"stats\">\n      <div class=\"stat-item\"><span class=\"dot dot-correct\"><\/span> \u2713 <strong id=\"cvs-sc\">0<\/strong><\/div>\n      <div class=\"stat-item\"><span class=\"dot dot-wrong\"><\/span> \u2717 <strong id=\"cvs-sw\">0<\/strong><\/div>\n      <div class=\"stat-item\"><span class=\"dot dot-rem\"><\/span> Left <strong id=\"cvs-sr\">15<\/strong><\/div>\n      <div class=\"sep\"><\/div>\n      <div class=\"stat-item\"><span class=\"dot dot-net\"><\/span> Net <strong id=\"cvs-sn\">0<\/strong><\/div>\n      <div class=\"stat-item\">Max <strong id=\"cvs-sm\">60<\/strong><\/div>\n    <\/div>\n  <\/div>\n\n  <div class=\"quiz-main\">\n    <div id=\"cvs-container\"><\/div>\n    <button class=\"submit-btn\" id=\"cvs-submit\">View Score \u2192<\/button>\n    <div class=\"score-panel\" id=\"cvs-score\"><\/div>\n  <\/div>\n<\/div>\n\n<script>\n(function () {\n\n  const Q = {\n    container : document.getElementById('cvs-container'),\n    submit    : document.getElementById('cvs-submit'),\n    score     : document.getElementById('cvs-score'),\n    progress  : document.getElementById('cvs-progress'),\n    sc: document.getElementById('cvs-sc'),\n    sw: document.getElementById('cvs-sw'),\n    sr: document.getElementById('cvs-sr'),\n    sn: document.getElementById('cvs-sn'),\n    sm: document.getElementById('cvs-sm')\n  };\n\n  const BANK = [\n    {\n      section: \"Vascular Disease\",\n      q: \"In hyaline arteriolosclerosis, the primary pathological change is:\",\n      correctAnswer: \"Homogeneous pink thickening of arteriolar walls due to plasma protein insudation and basement membrane reduplication\",\n      opts: [\n        \"Homogeneous pink thickening of arteriolar walls due to plasma protein insudation and basement membrane reduplication\",\n        \"Transmural necrosis with fibrinoid change and inflammatory infiltrate\",\n        \"Concentric onion-skin fibrosis of the arteriolar wall\",\n        \"Calcification confined to the tunica media without luminal narrowing\"\n      ],\n      exp: \"<strong>Hyaline arteriolosclerosis<\/strong>: benign form seen in elderly and diabetics. Plasma proteins leak into the wall \u2192 homogeneous eosinophilic (hyaline) thickening \u2192 luminal narrowing. <strong>Hyperplastic ('onion-skin') arteriolosclerosis<\/strong> (option C) is the malignant-hypertension variant \u2014 concentric smooth muscle lamination + fibrinoid necrosis (option B).\",\n      difficulty: \"medium\"\n    },\n    {\n      section: \"Vascular Disease\",\n      q: \"A 58-year-old with poorly controlled hypertension presents with sudden-onset severe headache and BP 220\/140. Renal biopsy shows 'flea-bitten' cortical petechiae. The arteriolar lesion responsible is:\",\n      correctAnswer: \"Hyperplastic arteriolosclerosis with fibrinoid necrosis\",\n      opts: [\n        \"Hyaline arteriolosclerosis\",\n        \"M\u00f6nckeberg medial sclerosis\",\n        \"Atherosclerosis with complicated plaque\",\n        \"Hyperplastic arteriolosclerosis with fibrinoid necrosis\"\n      ],\n      exp: \"<strong>Malignant hypertension<\/strong> \u2192 <strong>hyperplastic arteriolosclerosis<\/strong>: concentric onion-skin smooth muscle proliferation + <strong>fibrinoid necrosis<\/strong> of the arteriolar wall. The 'flea-bitten' kidney = petechial haemorrhages from ruptured arterioles. Hyaline change (A) is benign hypertension; M\u00f6nckeberg (B) does not narrow the lumen.\",\n      difficulty: \"medium\"\n    },\n    {\n      section: \"Atherosclerosis\",\n      q: \"The earliest grossly visible lesion in atherosclerosis, reversible and present even in children, is the:\",\n      correctAnswer: \"Fatty streak\",\n      opts: [\n        \"Fibrous plaque\",\n        \"Complicated plaque with calcification\",\n        \"Fatty streak\",\n        \"Gelatinous plaque\"\n      ],\n      exp: \"<strong>Fatty streak<\/strong>: flat, yellow intimal streak composed of lipid-laden foam cells (macrophages + some smooth muscle cells). Appears in aorta of children; does not obstruct flow; <em>potentially reversible<\/em>. Progresses over decades to fibrous plaque \u2192 complicated plaque (calcification, ulceration, thrombus, haemorrhage).\",\n      difficulty: \"easy\"\n    },\n    {\n      section: \"Atherosclerosis\",\n      q: \"Which complication of an atherosclerotic plaque is most directly responsible for acute myocardial infarction?\",\n      correctAnswer: \"Plaque rupture with superimposed thrombosis causing acute occlusion\",\n      opts: [\n        \"Plaque rupture with superimposed thrombosis causing acute occlusion\",\n        \"Slow progressive luminal narrowing beyond 70% stenosis\",\n        \"Calcification stiffening the vessel wall\",\n        \"Aneurysm formation distal to the plaque\"\n      ],\n      exp: \"<strong>Plaque rupture<\/strong> (or erosion) exposes subendothelial collagen and lipid core \u2192 platelet aggregation + coagulation cascade \u2192 <strong>occlusive thrombus<\/strong> \u2192 acute MI. Chronic \u226570% stenosis causes stable angina. Calcification increases stiffness but is not the acute trigger. Aneurysm is a different sequela.\",\n      difficulty: \"easy\"\n    },\n    {\n      section: \"Aneurysm & Dissection\",\n      q: \"A true aneurysm differs from a false aneurysm (pseudoaneurysm) in that:\",\n      correctAnswer: \"True aneurysms involve dilatation of all three wall layers; false aneurysms are contained haematomas outside the wall\",\n      opts: [\n        \"True aneurysms are always caused by infection\",\n        \"False aneurysms involve all three layers of the arterial wall\",\n        \"True aneurysms rupture more frequently than false aneurysms\",\n        \"True aneurysms involve dilatation of all three wall layers; false aneurysms are contained haematomas outside the wall\"\n      ],\n      exp: \"<strong>True aneurysm<\/strong>: dilatation involving all three layers (intima, media, adventitia) \u2014 e.g., atherosclerotic AAA, syphilitic. <strong>False (pseudo) aneurysm<\/strong>: breach of the wall \u2192 pulsating haematoma contained only by perivascular tissue\/clot \u2014 e.g., post-traumatic, post-anastomotic. Both can rupture; pseudoaneurysms are considered inherently unstable.\",\n      difficulty: \"medium\"\n    },\n    {\n      section: \"Vasculitis\",\n      q: \"Polyarteritis nodosa (PAN) characteristically:\",\n      correctAnswer: \"Affects medium-sized muscular arteries segmentally, sparing the lungs, and is p-ANCA negative\",\n      opts: [\n        \"Involves pulmonary vessels and is ANCA-positive in most cases\",\n        \"Affects medium-sized muscular arteries segmentally, sparing the lungs, and is p-ANCA negative\",\n        \"Causes granulomatous inflammation of large elastic arteries in young women\",\n        \"Is limited to the skin and peripheral nerves in elderly patients\"\n      ],\n      exp: \"<strong>PAN<\/strong>: necrotising vasculitis of <em>medium-sized muscular arteries<\/em> \u2014 segmental, skip lesions. Key feature: <strong>lung sparing<\/strong> (unlike ANCA-associated vasculitides). Associated with <em>hepatitis B<\/em> (30%). <strong>p-ANCA negative<\/strong>. Granulomatous large-vessel vasculitis = GCA (elderly) or Takayasu (young women). Lung + kidney involvement = Granulomatosis with polyangiitis (GPA, formerly Wegener's) \u2014 c-ANCA+.\",\n      difficulty: \"hard\"\n    },\n    {\n      section: \"Heart Failure\",\n      q: \"Right-sided heart failure most characteristically causes:\",\n      correctAnswer: \"Congestive hepatomegaly, dependent pitting oedema, and raised JVP\",\n      opts: [\n        \"Pulmonary oedema and orthopnoea\",\n        \"Paroxysmal nocturnal dyspnoea\",\n        \"Congestive hepatomegaly, dependent pitting oedema, and raised JVP\",\n        \"Cardiac asthma due to bronchospasm\"\n      ],\n      exp: \"<strong>Right heart failure<\/strong> \u2192 systemic venous congestion: <strong>congestive hepatomegaly<\/strong> (nutmeg liver \u2192 cardiac cirrhosis), <strong>peripheral oedema<\/strong> (ankles\/sacrum), raised JVP, ascites, splenomegaly. Pulmonary oedema (A, B, D) = <em>left<\/em> heart failure features. The distinction is clinically important and a common exam trap.\",\n      difficulty: \"easy\"\n    },\n    {\n      section: \"Ischaemic Heart Disease\",\n      q: \"On histology of a myocardial infarct at 3\u20135 days, the predominant inflammatory cell infiltrate is:\",\n      correctAnswer: \"Neutrophils undergoing lysis, being replaced by macrophages\",\n      opts: [\n        \"Neutrophils undergoing lysis, being replaced by macrophages\",\n        \"Lymphocytes and plasma cells\",\n        \"Eosinophils with multinucleated giant cells\",\n        \"Mast cells and basophils\"\n      ],\n      exp: \"MI histology timeline: <strong>0\u20134 hr<\/strong>: no LM change. <strong>4\u201312 hr<\/strong>: wavy fibres, coagulative necrosis. <strong>12\u201324 hr<\/strong>: neutrophil influx (peak). <strong>3\u20135 days<\/strong>: <strong>neutrophils lyse<\/strong>, macrophages arrive to phagocytose debris. <strong>1\u20132 wk<\/strong>: granulation tissue. <strong>6+ wk<\/strong>: dense fibrous scar. This timeline is high-yield for NEET-PG\/USMLE.\",\n      difficulty: \"hard\"\n    },\n    {\n      section: \"Ischaemic Heart Disease\",\n      q: \"Dressler syndrome, occurring 2\u201310 weeks after MI, is best explained by:\",\n      correctAnswer: \"Autoimmune pericarditis triggered by release of cardiac antigens\",\n      opts: [\n        \"Reinfarction due to stent restenosis\",\n        \"Ventricular free wall rupture leading to haemopericardium\",\n        \"Extension of myocardial necrosis into the pericardium\",\n        \"Autoimmune pericarditis triggered by release of cardiac antigens\"\n      ],\n      exp: \"<strong>Dressler syndrome<\/strong> (post-MI pericarditis): delayed (2\u201310 weeks), due to <strong>autoimmune reaction<\/strong> against released myocardial antigens. Features: fever, pleuritic chest pain, pericardial friction rub, elevated ESR. Treat with NSAIDs\/aspirin. Not recurrence of ischaemia. Haemopericardium (B) from free wall rupture occurs earlier (3\u20135 days).\",\n      difficulty: \"medium\"\n    },\n    {\n      section: \"Ischaemic Heart Disease\",\n      q: \"Chronic ischaemic heart disease with heart failure is characterised pathologically by:\",\n      correctAnswer: \"Progressive myocyte loss with multifocal interstitial fibrosis and compensatory hypertrophy\",\n      opts: [\n        \"Large transmural scar replacing the full thickness of the ventricular wall at one site\",\n        \"Diffuse hypertrophy without any fibrosis\",\n        \"Progressive myocyte loss with multifocal interstitial fibrosis and compensatory hypertrophy\",\n        \"Amyloid deposition replacing the myocardium uniformly\"\n      ],\n      exp: \"<strong>Chronic IHD<\/strong>: not a single large infarct, but <em>accumulation<\/em> of small ischaemic injuries \u2192 <strong>multifocal fibrosis<\/strong>, myocyte loss, and compensatory hypertrophy of surviving fibres. The heart is enlarged but function deteriorates progressively \u2192 heart failure. Distinct from acute MI (single event) and from dilated cardiomyopathy (no ischaemic basis).\",\n      difficulty: \"medium\"\n    },\n    {\n      section: \"Valvular Heart Disease\",\n      q: \"Rheumatic mitral stenosis results primarily from:\",\n      correctAnswer: \"Repeated episodes of rheumatic carditis causing leaflet thickening, commissural fusion, and chordal shortening\",\n      opts: [\n        \"Repeated episodes of rheumatic carditis causing leaflet thickening, commissural fusion, and chordal shortening\",\n        \"Calcification of the mitral annulus in elderly patients\",\n        \"Infective endocarditis destroying the leaflet tissue\",\n        \"Myxomatous degeneration with leaflet prolapse\"\n      ],\n      exp: \"<strong>Rheumatic mitral stenosis<\/strong>: sequela of group A strep pharyngitis \u2192 molecular mimicry \u2192 autoimmune carditis. Repeated attacks \u2192 fibrous thickening of leaflets, commissural fusion, chordal shortening\/fusion \u2192 'fish-mouth' orifice. <em>Most common cause of MS worldwide.<\/em> Calcific AS (B) affects the aortic valve in elderly; myxomatous change (D) causes <em>prolapse<\/em>, not stenosis.\",\n      difficulty: \"medium\"\n    },\n    {\n      section: \"Valvular Heart Disease\",\n      q: \"Libman-Sacks endocarditis is associated with:\",\n      correctAnswer: \"Systemic lupus erythematosus, with small sterile vegetations on both surfaces of the mitral valve\",\n      opts: [\n        \"IV drug use and Staphylococcus aureus\",\n        \"Rheumatic fever and group A Streptococcus\",\n        \"Chronic debilitating illness and hypercoagulable state (NBTE)\",\n        \"Systemic lupus erythematosus, with small sterile vegetations on both surfaces of the mitral valve\"\n      ],\n      exp: \"<strong>Libman-Sacks endocarditis<\/strong>: non-infective, associated with <strong>SLE<\/strong>. Small, warty, sterile vegetations on <em>both surfaces<\/em> of the mitral (and tricuspid) leaflets \u2014 a distinguishing feature from NBTE (line of closure only) and rheumatic (atrial surface). Antiphospholipid antibodies contribute to the procoagulant state.\",\n      difficulty: \"hard\"\n    },\n    {\n      section: \"Cardiomyopathies\",\n      q: \"Arrhythmogenic right ventricular cardiomyopathy (ARVC) is characterised by:\",\n      correctAnswer: \"Fibrofatty replacement of right ventricular myocardium causing ventricular arrhythmias\",\n      opts: [\n        \"Symmetric biventricular dilatation with reduced EF\",\n        \"Asymmetric septal hypertrophy with dynamic LVOTO\",\n        \"Fibrofatty replacement of right ventricular myocardium causing ventricular arrhythmias\",\n        \"Endomyocardial fibrosis obliterating the RV apex\"\n      ],\n      exp: \"<strong>ARVC<\/strong>: autosomal dominant desmosome mutations (plakophilin-2, desmoplakin). RV myocytes die and are replaced by <strong>fibrofatty tissue<\/strong> \u2192 electrical instability \u2192 ventricular tachycardia\/fibrillation \u2192 sudden cardiac death in young athletes. Epsilon wave on ECG. <em>Not<\/em> the same as DCM (A) or HCM (B). Endomyocardial fibrosis (D) is a restrictive pattern, tropical aetiology.\",\n      difficulty: \"hard\"\n    },\n    {\n      section: \"Cardiomyopathies\",\n      q: \"Restrictive cardiomyopathy most commonly produces:\",\n      correctAnswer: \"Diastolic dysfunction with normal or near-normal systolic function and small, stiff ventricles\",\n      opts: [\n        \"Systolic dysfunction with markedly reduced ejection fraction\",\n        \"Diastolic dysfunction with normal or near-normal systolic function and small, stiff ventricles\",\n        \"Dynamic left ventricular outflow tract obstruction\",\n        \"Massive biventricular dilatation with mural thrombi\"\n      ],\n      exp: \"<strong>Restrictive cardiomyopathy<\/strong>: stiff, non-compliant ventricles (due to amyloidosis, haemochromatosis, sarcoidosis, endomyocardial fibrosis) \u2192 impaired diastolic filling \u2192 <strong>diastolic dysfunction<\/strong>. Systolic function (EF) is <em>preserved<\/em> initially. Small or normal ventricular size. Raised filling pressures mimic constrictive pericarditis. Compare: DCM = systolic dysfunction + dilated chambers.\",\n      difficulty: \"medium\"\n    },\n    {\n      section: \"Hypertensive Heart Disease\",\n      q: \"The earliest and most characteristic morphological finding in systemic (left-sided) hypertensive heart disease is:\",\n      correctAnswer: \"Concentric left ventricular hypertrophy with a normal or reduced cavity size\",\n      opts: [\n        \"Concentric left ventricular hypertrophy with a normal or reduced cavity size\",\n        \"Eccentric LV dilatation with thin walls\",\n        \"Biventricular dilatation with mural thrombi\",\n        \"Mitral valve prolapse with leaflet redundancy\"\n      ],\n      exp: \"<strong>Systemic hypertensive heart disease<\/strong>: chronic pressure overload \u2192 sarcomere addition in <em>parallel<\/em> \u2192 <strong>concentric LV hypertrophy<\/strong> (thick walls, normal\/small cavity). LV weight may exceed 500 g (normal ~250\u2013300 g). Predisposes to diastolic dysfunction, arrhythmias, and sudden death. Eccentric dilatation (B) is volume overload or end-stage decompensation.\",\n      difficulty: \"easy\"\n    }\n  ];\n\n  function shuffle(arr) {\n    const a = [...arr];\n    for (let i = a.length - 1; i > 0; i--) {\n      const j = Math.floor(Math.random() * (i + 1));\n      [a[i], a[j]] = [a[j], a[i]];\n    }\n    return a;\n  }\n\n  let session = [], answered = [], total = 0;\n\n  function prepareSession() {\n    session = shuffle(BANK).map(q => {\n      const opts = shuffle(q.opts);\n      return { ...q, opts, ansIdx: opts.indexOf(q.correctAnswer) };\n    });\n    answered = new Array(session.length).fill(null);\n    total = 0;\n    Q.sn.textContent = '0';\n    Q.sm.textContent = session.length * 4;\n  }\n\n  function diffBadge(d) {\n    return d ? `<span class=\"diff diff-${d}\">${d}<\/span>` : '';\n  }\n\n  function buildQuiz() {\n    Q.container.innerHTML = '';\n    Q.sr.textContent = session.length;\n\n    session.forEach((q, i) => {\n      const card = document.createElement('div');\n      card.className = 'q-card';\n\n      const letters = ['A','B','C','D'];\n      const optsHTML = q.opts.map((opt, j) => `\n        <div class=\"opt\" id=\"cvs-o-${i}-${j}\">\n          <span class=\"opt-letter\">${letters[j]}<\/span>\n          <span>${opt}<\/span>\n        <\/div>`).join('');\n\n      card.innerHTML = `\n        <div class=\"q-head\">\n          <div class=\"q-num\" id=\"cvs-n-${i}\">${i + 1}<\/div>\n          <div class=\"q-text\">${q.q}<\/div>\n        <\/div>\n        <div class=\"options\">${optsHTML}<\/div>\n        <div class=\"explanation\" id=\"cvs-e-${i}\">\n          <div class=\"exp-label\">Explanation ${diffBadge(q.difficulty)}<\/div>\n          ${q.exp}\n        <\/div>`;\n\n      Q.container.appendChild(card);\n      q.opts.forEach((_, j) => {\n        document.getElementById(`cvs-o-${i}-${j}`)\n          .addEventListener('click', () => answer(i, j));\n      });\n    });\n\n    Q.submit.classList.remove('visible');\n  }\n\n  function answer(qi, oi) {\n    if (answered[qi] !== null) return;\n    const q = session[qi];\n    const ok = oi === q.ansIdx;\n    answered[qi] = ok ? 'correct' : 'wrong';\n    total++;\n\n    q.opts.forEach((_, j) => {\n      const el = document.getElementById(`cvs-o-${qi}-${j}`);\n      if (j === q.ansIdx)       el.classList.add('correct');\n      else if (j === oi && !ok) el.classList.add('wrong');\n      else                      el.classList.add('dimmed');\n    });\n\n    document.getElementById(`cvs-e-${qi}`).classList.add('show');\n    document.getElementById(`cvs-n-${qi}`).classList.add(ok ? 'answered-c' : 'answered-w');\n\n    updateStats();\n    updateProgress();\n    if (total === session.length) Q.submit.classList.add('visible');\n  }\n\n  function updateStats() {\n    const c = answered.filter(a => a === 'correct').length;\n    const w = answered.filter(a => a === 'wrong').length;\n    const r = answered.filter(a => a === null).length;\n    const n = (c * 4) - w;\n    Q.sc.textContent = c;\n    Q.sw.textContent = w;\n    Q.sr.textContent = r;\n    Q.sn.textContent = n >= 0 ? '+' + n : n;\n    Q.sm.textContent = session.length * 4;\n  }\n\n  function updateProgress() {\n    Q.progress.style.width = (total \/ session.length * 100) + '%';\n  }\n\n  function showScore() {\n    const c  = answered.filter(a => a === 'correct').length;\n    const w  = answered.filter(a => a === 'wrong').length;\n    const sk = answered.filter(a => a === null).length;\n    const mx = session.length * 4;\n    const ns = (c * 4) - w;\n    const pc = Math.round((ns \/ mx) * 100);\n    const bw = Math.max(0, Math.min(100, pc));\n    const msg = pc >= 85 ? 'Excellent grasp of cardiovascular pathology!' :\n                pc >= 65 ? 'Good foundation \u2014 review the explanations above.' :\n                pc >= 50 ? 'Revisit key concepts; focus on weak sections.' :\n                pc >= 0  ? 'Dedicated revision needed \u2014 use explanations as study notes.' :\n                           'Negative net score \u2014 in NEET-PG, blind guessing hurts.';\n\n    Q.score.innerHTML = `\n      <h2>Quiz Complete<\/h2>\n      <div class=\"score-num\">${ns < 0 ? ns : '+' + ns}<\/div>\n      <div class=\"score-denom\">Net Score &nbsp;|&nbsp; Max Marks: ${mx}<\/div>\n      <div class=\"grade-bar\"><div class=\"grade-fill\" style=\"width:${bw}%\"><\/div><\/div>\n      <div class=\"score-breakdown\">\n        <span><strong>${c}<\/strong><br>Correct<br><small>(+${c * 4})<\/small><\/span>\n        <span><strong>${w}<\/strong><br>Wrong<br><small>(\u2212${w})<\/small><\/span>\n        <span><strong>${sk}<\/strong><br>Skipped<br><small>(0)<\/small><\/span>\n        <span><strong>${Math.max(0, pc)}%<\/strong><br>Score%<\/span>\n      <\/div>\n      <div class=\"score-formula\">\n        Net = (${c} \u00d7 4) \u2212 ${w} = <strong>${ns}<\/strong>\n        &nbsp;|&nbsp; ${ns} \u00f7 ${mx} \u00d7 100 = <strong>${pc}%<\/strong>\n      <\/div>\n      <div class=\"score-msg\">${msg}<\/div>\n      <button class=\"btn-retry\" id=\"cvs-retry\">\u21ba Reshuffle &amp; Retry<\/button>`;\n\n    Q.score.style.display = 'block';\n    Q.submit.classList.remove('visible');\n    Q.score.scrollIntoView({ behavior: 'smooth' });\n    document.getElementById('cvs-retry').addEventListener('click', retryQuiz);\n  }\n\n  function retryQuiz() {\n    Q.score.style.display = 'none';\n    prepareSession();\n    updateStats();\n    updateProgress();\n    buildQuiz();\n    window.scrollTo({ top: 0, behavior: 'smooth' });\n  }\n\n  Q.submit.addEventListener('click', showScore);\n  prepareSession();\n  buildQuiz();\n  updateStats();\n\n  new IntersectionObserver(\n    ([e]) => document.getElementById('cvs-status-bar')\n                      .classList.toggle('visible', !e.isIntersecting),\n    { threshold: 0 }\n  ).observe(document.getElementById('cvs-sentinel'));\n\n})();\n<\/script>\n<\/div>\n\n\n","protected":false},"excerpt":{"rendered":"<p>\u2713 0 \u2717 0 Left 15 Net 0 Max 60 View Score \u2192<\/p>\n","protected":false},"author":1,"featured_media":0,"comment_status":"open","ping_status":"open","sticky":false,"template":"","format":"standard","meta":{"neve_meta_sidebar":"","neve_meta_container":"","neve_meta_enable_content_width":"","neve_meta_content_width":0,"neve_meta_title_alignment":"","neve_meta_author_avatar":"","neve_post_elements_order":"","neve_meta_disable_header":"","neve_meta_disable_footer":"","neve_meta_disable_title":"","footnotes":""},"categories":[24],"tags":[],"class_list":["post-36406","post","type-post","status-publish","format-standard","hentry","category-neet-pg"],"yoast_head":"<!-- This site is optimized with the Yoast SEO plugin v27.3 - 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